prof. Mgr. Vítězslav Bryja, Ph.D.
Department head, Department of Experimental Biology
correspondence Address:
Kotlářská 267/2, 611 37 Brno
office: bldg. D36/112
Kamenice 753/5
625 00 Brno
phone: | +420 549 49 3291 |
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e‑mail: |
social and academic networks: |
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Total number of publications: 218
2013
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Wnt5a cooperates with canonical Wnts to generate midbrain dopaminergic neurons in vivo and in stem cells.
Proceedings of the National Academy of Sciences of the United States of America, year: 2013, volume: 110, edition: 7, DOI
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Wnt-PCP pathway driving pathogenesis of chronic lymphocytic leukemia participates on normal B-cell development
Year: 2013, type: Conference abstract
2012
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19th Acta Physiologica international symposium WNT Signaling in Physiology and Disease'- a long way from developmental biology to physiology
Year: 2012, type: Article in Periodical (without peer review)
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Exome capture reveals ZNF423 and CEP164 mutations, linking renal ciliopathies to DNA damage response signaling
Cell, year: 2012, volume: 150, edition: 3, DOI
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Receptor tyrosine kinases activate canonical WNT/beta catenin signaling via MAP kinase/LRP6 pathway and direct beta-Catenin phosphorylation
Plos One, year: 2012, volume: 7, edition: 4, DOI
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SFRP1 and SFRP2 dose-dependently regulate midbrain dopamine neuron development in vivo and in embryonic stem cells.
Stem Cells, year: 2012, volume: 30, edition: 5, DOI
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Způsob stanovení závažnosti a prognózy B-buněčné chronické lymfocytární leukémie
Publisher: , state: Czech Republic, patent's number: 303555, year: 2012
2011
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Amer1/WTX couples Wnt-induced formation of PtdIns(4,5)P(2) to LRP6 phosphorylation.
The EMBO journal, year: 2011, volume: 30, edition: 8, DOI
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Gene expression changes in human prostate carcinoma cells exposed to genotoxic and nongenotoxic aryl hydrocarbon receptor ligands
TOXICOLOGY LETTERS, year: 2011, volume: 206, edition: 2, DOI
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Mitogen-Activated Protein Kinases Promote WNT/beta-Catenin Signaling via Phosphorylation of LRP6
Molecular and cellular biology, year: 2011, volume: 31, edition: 1, DOI